Elicit: Predictive Markers for NSAA/6MWT in Dystrophinopathies (public)

Do micro-dystrophin expression and CK decline at 12 months predict NSAA/6MWT at 24–36 months?

Abstract

Micro‐dystrophin expression measured within the first 12 months is reported to parallel later ambulatory function in Duchenne muscular dystrophy. Studies document that robust micro‐dystrophin induction—ranging from approximately 24% to nearly 40% of normal levels measured at 12 weeks to 12 months—is accompanied by North Star Ambulatory Assessment (NSAA) improvements of +1.3 to +7.0 points at 48 weeks to 2 years. In several trials, declines in creatine kinase (e.g., a reduction of about 4,344 units/L) track with such functional gains, and interventions have produced 6‐minute walk test improvements of up to +162 meters.

No study directly models micro‐dystrophin expression or creatine kinase decline as individual predictors of later NSAA or 6‐minute walk test performance, nor do any combine these markers in a single analysis. Instead, group‐level associations and temporal trends consistently link early biomarker shifts with later stabilization or improvement in ambulation outcomes.

Methods

We analyzed 40 sources from an initial pool of 997, using 7 screening criteria. Each paper was reviewed for 6 key aspects that mattered most to the research question.

Screening Criteria

Results

Characteristics of Included Studies

Study Study Design Patient Population Measurement Methods Primary Outcomes
Mendell et al., 2021a Open-label study 4 ambulatory boys, 4–7 years, Duchenne muscular dystrophy Immunofluorescence, Western blot (micro-dystrophin), North Star Ambulatory Assessment Safety, micro-dystrophin expression, North Star Ambulatory Assessment
Goemans et al., 2016 Open-label extension 12 boys, mean 9.5 years, Duchenne muscular dystrophy, exon 51 amenable Muscle biopsy (dystrophin), 6-minute walk test Long-term efficacy, safety, pharmacokinetics
Pascual-Morena et al., 2020 Systematic review/meta-analysis Children/adolescents with Duchenne muscular dystrophy Pooled clinical trial data Functional outcomes, dystrophin expression
Rao et al., 2021 Prospective cohort, open-label Duchenne muscular dystrophy patients, age not reported Western blot (micro-dystrophin), functional tests Safety, micro-dystrophin, function
Goemans et al., 2015a Prospective cohort 269 boys, 3–18 years, Duchenne muscular dystrophy 6-minute walk test, North Star Ambulatory Assessment, timed function tests Natural history, biomarkers
Mendell et al., 2021b Randomized controlled trial, crossover, open-label extension 41 boys, 4–7 years, Duchenne muscular dystrophy Western blot (micro-dystrophin), North Star Ambulatory Assessment Safety, efficacy
Mendell et al., 2013 Randomized controlled trial, open-label extension 12 boys, 7–13 years, exon 51 amenable 6-minute walk test Efficacy, safety
Mendell et al., 2014 Prospective cohort, randomized controlled trial elements 12 boys, 7–13 years, exon 51 amenable 6-minute walk test, pulmonary function test Efficacy, safety
Mendell et al., 2023a Randomized controlled trial, crossover, open-label extension 41 boys, ≥4–<8 years, Duchenne muscular dystrophy Western blot (micro-dystrophin), North Star Ambulatory Assessment Safety, efficacy
Zaidman et al., 2021 Open-label, Phase 1b 20 boys, 4–7 years, Duchenne muscular dystrophy Western blot, immunofluorescence (micro-dystrophin), North Star Ambulatory Assessment Expression, safety

Predictive Markers at 12 Months

Functional Outcomes at 24–36 Months

Key Findings

Synthesis and Limitations

The available evidence indicates that micro-dystrophin expression and creatine kinase decline at 12 months are associated with stabilization or improvement in North Star Ambulatory Assessment and 6-minute walk test at 24–36 months in treated Duchenne muscular dystrophy populations. However, the absence of individual-level predictive modeling limits the conclusions.